JANUARY 6, 2004. Below I have reprinted a fascinating excerpt from 1952 testimony given by Dr. Ralph R Scobey before a US House of Representatives Committee looking into the problem of toxins in the food supply.
Dr. Scobey begins by citing historical references on polio which indicate that a similar (or identical) condition has been caused by poisons ingested by humans.
The doctor aims his attack on researchers who are insisting that polio is infectious and is caused by a germ. He mentions that, in all situations of any illness, deluded researchers believe a widespread number of cases constitutes solid evidence that some communicable germ is the cause---when in fact, this is not necessarily the case.
From our point in time, the doctor's remarks seem fatuous because we believe the situation is settled: polio IS caused by a virus and it WAS stopped in its tracks by a vaccine which contained the poliovirus and made the body produce antibodies that would defeat polio.
In exactly the same way, we believe that a prion causes mad cow and HIV causes AIDS and a newly discovered coronavirus causes SARS.
Yes, well, scientists were at one time positive that a condition called pellagra was an infectious disease caused by a germ---until they were forced to admit that it was a severe deficiency of niacin.
Dr. Scobey, in 1952, was pointing out that there was no real evidence that polio was one condition with one infectious cause.
Over and over again, we are bamboozled by professional disease propaganda that presents illness as contagious and germ-driven.
Years after Dr. Scobey rendered his testimony, it was revealed that polio had actually been on its way out before the vaccine was introduced, and the vaccine did nothing to change that trend. It was also shown that, by a clever change in definition AFTER the vaccine was introduced, the case numbers seemed to drop, lending false strength to the assertion that the vaccine was successful.
The word DISEASE is a befuddling one. We automatically assume it means a condition caused by a germ. But its close cousin, ILLNESS, is a little more descriptive. A person can be ill without a germ. A pesticide, for example, can make him ill. But by invoking the word DISEASE to cover almost every situation in which people fall ill, we are persuaded to think a germ is the reason.
Here is a telling excerpt from Dr. Scobey's THE POISON CAUSE OF POLIOMYELITIS AND OBSTRUCTIONS TO ITS INVESTIGATION, given to the House Select Committee to Investigate the Use of Chemicals in Food Products:
It is obvious that in the study of poliomyelitis every possible cause, including the possibility of poisoning, should be investigated.Since 1908 -- for 44 years -- poliomyelitis research has been predominantly directed along only one line of investigation, i.e., the infectious theory. This single line of study, precluding other possibilities, including the poison cause of the disease, has resulted from two factors, (1) The Public Health Law93, and (2) the insistence, based entirely on animal experiments, that poliomyelitis is caused by a virus.
1. The Public Health Law. The inclusion of poliomyelitis in the Public Health Law as a communicable, infectious disease dates back to the early part of the 20th Century. At that time many diseases, now known to be neither communicable nor infectious, were considered to be caused by an infectious agent simply because they occurred in epidemics. The general attitude of that period is expressed by Sachs94 (1911) in his statement: "In general, the epidemic occurrence of any disease is sufficient to prove its infectious or contagious character." The vitamin deficiency diseases, beriberi and pellagra, are outstanding examples of epidemic diseases that were formerly considered to be infectious and communicable according to the logic employed by Sachs. In fact, we find pellagra incorporated into the Public Health Law as a communicable disease in the State of Pennsylvania in the following rule and regulation adopted January 5, MEDICAL MIND SET 1910:
"That all physicians practicing within the limits of the state shall make immediate report of each and every case of uncinariasis duodenalis (hookworm disease) and pellagra and anterior poliomyelitis (infantile paralysis) occurring in their practice in the same manner that other communicable diseases are now by law and by rule and regulation of the State Department of Health reported to the health authorities." A State Health Officer95 recently wrote to me as follows: "I think all of us will agree with you that in the past, as is still probably true, public health rules and regulations and sometimes even public health laws, were influenced too much by what we did not know rather than by what we did know. This was probably an acceptable line of reasoning in the past, but with increasing public health education and greater understanding and cooperation from citizens, this justification becomes less acceptable."The fact that an extensive epidemic of poliomyelitis was prevailing in the states of New York and Massachusetts in 1907, aroused the suspicion that the disease was infectious and communicable; it was therefore incorporated into the Public Health Law as such. However, conclusive evidence of contagiousness was not established during that epidemic nor in subsequent ones. Moreover, [the same lack of evidence was revealed] during the greatest epidemic of poliomyelitis in recorded history, as shown by the records of the U.S. Public Health Service and the New York State Department of Health. Time Magazine, commenting on these surveys, points out how, when and where people catch polio remained a mystery. In addition to the failure to prove contagiousness of human poliomyelitis, it has likewise been impossible to prove contagiousness of poliomyelitis in experimental animals. This fact will be considered in detail later.
As a result of the inclusion of poliomyelitis in the Public Health Law as a contagious, communicable or infectious disease, investigations regarding it are almost exclusively in the hands of specialists in virology and public health. The country doctor, general practitioner, and clinician have little or no opportunity to participate in poliomyelitis research under these circumstances. Yet, Dr. W. Ritchie Russell97 of the Department of Neurology, United Oxford Hospitals, Oxford, England stated in 1950: "Clinical research into this disease is so much neglected that there are exciting discoveries waiting for anyone with time to give to this type of investigation."
... All advances in medicine do not result from laboratory experiments. Any doctor in any community, however, small, and however limited his opportunities, may make a fundamental discovery, but he must be given the opportunity to participate in the program and his observations and deductions must be given adequate consideration.
2. Virus Research. The more or less general acceptance of the idea that poliomyelitis is caused by a virus arose from experimental animal studies by Landsteiner98 (1908) in Austria, and Flexner and Lewis99 (1909) in the United States. These experiments showed that a substance obtained from poliomyelitis victims could produce a paralytic disease when administered to experimental animals. It has been assumed, as a result of these experiments, that an exogenous [originating from without] virus is the cause of human poliomyelitis. Dr. Harold L. Amoss100 stated in 1928: "By reason of the parallelism of the human and experimental disease it is believed that inferences drawn from experiments with monkeys may be accepted with a certain degree of safety as applicable to the solution of problems in connection with human cases." The portal of entry of the so-called virus of poliomyelitis into the human body never has been established. The question of the portal of entry is summed up in the published reports of the International Poliomyelitis Congress that was held in New York City in 1948. The Modulator stated: "We do not know too much about the portal of entry in human beings," and Dr. John R. Paul, of Yale University, stated: "I would say we do not know the portal of entry in human beings."
It was mentioned in the foregoing that human poliomyelitis has not been shown conclusively to be a contagious disease. Neither has the experimental animal disease, produced by the so-called poliomyelitis virus, been shown to be communicable. Rosenau102 (1921) stated: "Monkeys have so far never been known to contract the disease "spontaneously" even though they are kept in intimate association with infected monkeys." Twenty years later (1941), Dr. John A. Toomey103, a poliomyelitis authority, stated: "No animal gets the disease from another no matter how intimately exposed."
It is extremely difficult to understand how a human can contract poliomyelitis from another individual through dissemination of a virus by contact, carriers, excrement, unclean hands, unwashed fruits and vegetables, flies, etc. when a healthy animal in the same cage with an "infected" animal, exposed to all of these natural factors, remains unaffected. It appears obvious, therefore, that communicability should have been established conclusively both in humans and in experimental animals before poliomyelitis was incorporated into the Public Health Law as a communicable disease.
Some investigators have as a matter of fact pointed out that human poliomyelitis and the disease produced in experimental animals from human material, etc. are not the same disease. Toomey104 (1935), for example, stated that intranasal and intracerebral inoculation of poliomyelitis virus in the monkey does not produce the same disease that is seen in man. Dr. Claus W. Jungebut, a well-known bacteriologist who has worked on the poliomyelitis problem for many years, recently stated (1950): "Whatever the final answer may be, it seems a reasonable statement at this time that the highly specialized, neurotropically fixed virus, which has been maintained in the past by intracerebral passage in rhesus monkeys, is more likely a laboratory artefact than the agent which causes the natural disease in man." The logical conclusion appears to be, therefore, that the laboratory experiments with the so-called virus of poliomyelitis are merely of academic interest and have no practical application to human poliomyelitis.
For almost half a century poliomyelitis investigations have been directed towards a supposed exogenous virus that enters the human body to cause the disease. The manner in which the Public Health Law is now stated imposes only this type of investigation. No intensive studies have been made, on the other hand, to determine whether or not the so-called virus of poliomyelitis is [rather] an autochthonous chemical substance that does not enter the human body at all, but simply results from an exogenous factor or factors, for example, a food poison. Analogous reactions are well-known as illustrated by the production of experimental sarcomas by indol, arsenic, tar, etc. and which have been transmitted by Berkefeld filtrates.
The discovery in recent years of the so-called Coxsackie virus has tended to further confuse the entire poliomyelitis problem. Hoyne107 (1951), for example, states that the announcement of this discovery "is accompanied by some feeling of dismay... In view of the foregoing announcement it seems that trained investigators have added one more problem to the nebulous conditions enveloping poliomyelitis. One might also be tempted to make the statement that the more we learn about poliomyelitis, the less we know." Hoyne's statement applies obviously to the confusion that has arisen from exclusive virus studies in poliomyelitis. A Lancet editorial108 (1951) also indicates the complexity of the problem brought about by the discovery of the Coxsackie virus, as follows: "A crop of new snags is coming along as every week brings new tidings of the Coxsackie viruses."
Many diseases have been considered to be caused by viruses but virus studies constitute only a portion of the investigations intended to determine the cause of the disease. Poliomyelitis investigations, on the other hand, have been confined exclusively to virus studies. Because of this situation and the Public Health Law, those who maintain other opinions, including those concerning poisons as the cause of poliomyelitis, can neither obtain funds from any source for research nor cooperation for investigating their ideas. Reappraisal and investigation of all theories, infectious and non-infectious, are imperative.
End of excerpt
It would be interesting to document the rise of paralytic conditions along with the introduction of more vaccines and pesticides into the general population in the early 20th century. Or for example, what about the introduction of fluorides into US drinking water, starting in 1950? Paralysis is one long-term potential effect of fluoride use at low levels---what was the level and the form of fluoride introduced in US in 1950? Was it high? Was it in a particularly toxic form? Was there effluent from the Manhattan Project, where radioactive material and fluorides were combined?
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